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Effect of bone loss on the fracture resistance of narrow dental implants after implantoplasty. An in vitro study

2021-09Journal article Open access
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dc.contributor.authorLeitão-Almeida, Bruno
dc.contributor.authorCamps-Font, Octavi
dc.contributor.authorCorreia, André
dc.contributor.authorMir-Mari, Javier
dc.contributor.authorFigueiredo, Rui
dc.contributor.authorValmaseda-Castellon, Eduard
dc.date.accessioned2021-09-13T08:43:08Z
dc.date.available2021-09-13T08:43:08Z
dc.date.issued2021-09
dc.description.abstractBackground: Implantoplasty (IP) involves polishing of the exposed surface of implants affected by peri-implantitis (PI). A study was made to determine whether the degree of bone loss influences the fracture resistance of implants with or without IP. Material and Methods: An in vitro study was carried out on 32 narrow (3.5 mm) dental implants with a rough surface and external hexagonal connection. Implantoplasty was performed in half of the implants of the sample. Both the IP and control implants were divided into two subgroups according to the amount of bone loss (3 mm or 7.5 mm). Standardized radiographic assessment of implant width was performed using specific software. The main outcome variable was the maximum compression force (F-max) of implants when subjected to static resistance to fracture tests. Implant fractures were subsequently analyzed by scanning electron microscopy. A descriptive and bivariate analysis of the data was performed. Results: Significant changes in implant width were observed after IP (p<0.05). No significant differences between IP and control implants were recorded in terms of the F-max values in the two bone loss subgroups (3 mm: control 854.37N +/- 195.08 vs. IP 752.12N +/- 186.13;p =0.302, and 7.5 mm: control 548.82N +/- 80.02 vs. IP 593.69N +/- 111.07; p-0.370). Greater bone loss was associated to a decrease in F-max which proved significant for the control implants (p=0.001). Fractures were more frequently located in the platform (n=13). Conclusions: Implants with more apical bone levels appear to be more susceptible to fracture. On the other hand, IP does not seem to significantly decrease the fracture resistance of narrow (3.5 mm) platform dental implants with external hexagonal connections. The fact that most fractures occur in the platform area indicates that the latter is exposed to more mechanical stress.pt_PT
dc.description.versioninfo:eu-repo/semantics/publishedVersionpt_PT
dc.identifier.doi10.4317/medoral.24624pt_PT
dc.identifier.eid85114249830
dc.identifier.issn1698-6946
dc.identifier.pmcPMC8412446
dc.identifier.pmid34162823
dc.identifier.urihttp://hdl.handle.net/10400.14/34757
dc.identifier.wos000691774300010
dc.language.isoengpt_PT
dc.peerreviewedyespt_PT
dc.subjectPeri-implantitispt_PT
dc.subjectDental implantspt_PT
dc.subjectCompressive strengthpt_PT
dc.subjectTitaniumpt_PT
dc.subjectImplantoplastypt_PT
dc.titleEffect of bone loss on the fracture resistance of narrow dental implants after implantoplasty. An in vitro studypt_PT
dc.typejournal article
dspace.entity.typePublication
oaire.citation.endPageE618pt_PT
oaire.citation.issue5pt_PT
oaire.citation.startPageE611pt_PT
oaire.citation.titleMedicina Oral, Patologia Oral y Cirugia Bucalpt_PT
oaire.citation.volume26pt_PT
person.familyNameAlmeida
person.familyNameCamps Font
person.familyNameCorreia
person.familyNameFigueiredo
person.familyNameValmaseda-Castellon
person.givenNameBruno
person.givenNameOctavi
person.givenNameAndre
person.givenNameRui
person.givenNameEduard
person.identifierR-000-NAN
person.identifier.ciencia-idDC19-CAE4-44C1
person.identifier.orcid0000-0001-6307-5585
person.identifier.orcid0000-0001-9991-2226
person.identifier.orcid0000-0002-0119-9790
person.identifier.orcid0000-0002-2122-6530
person.identifier.orcid0000-0001-9669-3187
person.identifier.ridA-5330-2010
person.identifier.ridC-2428-2008
person.identifier.scopus-author-id56681218200
person.identifier.scopus-author-id55418261400
person.identifier.scopus-author-id23004145100
person.identifier.scopus-author-id6602100038
rcaap.rightsopenAccesspt_PT
rcaap.typearticlept_PT
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relation.isAuthorOfPublication.latestForDiscovery68ea108f-e3d6-4232-a07b-a1c9a8843db4

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